Preliminary reports show conolidine may possibly inhibit specific ion channels, decreasing neuronal excitability and limiting agony signals. This system is particularly suitable in neuropathic pain, exactly where abnormal signaling causes persistent discomfort. Moreover, conolidine appears to impact G protein-coupled receptor (GPCR) pathways integral to agony perception. Computational... https://josephg158roi7.wikicommunications.com/user