Max in WT synaptoneurosomes, suggesting that Src signaling may very well be downregulated in KI synapses. Then again, our power to rescue SERT function in KI midbrain synaptoneurosomes by the inhibition of FAK indicates elevated FAK signaling downstream in the Pro32Pro33 mutant, as verified by enhanced pFAK localization in five-HT https://woodyn874nbt1.ouyawiki.com/user